Modulation of sestrin confers protection to Cr (VI) induced neuronal cell death in Drosophila melanogaster
P Singh, DK Chowdhuri - Chemosphere, 2018 - Elsevier
Increased oxidative stress is one of the major causes of hexavalent chromium [Cr (VI)], a
heavy metal with diverse applications and environmental presence, induced neuronal
adversities in exposed organism including Drosophila. Sestrin (sesn), an oxidative stress
responsive gene, emerges as a novel player in the management of oxidative stress
response. It is reported to be regulated by Target of rapamycin (TOR) and the former
regulates autophagy and plays an important role in the prevention of neurodegeneration …
heavy metal with diverse applications and environmental presence, induced neuronal
adversities in exposed organism including Drosophila. Sestrin (sesn), an oxidative stress
responsive gene, emerges as a novel player in the management of oxidative stress
response. It is reported to be regulated by Target of rapamycin (TOR) and the former
regulates autophagy and plays an important role in the prevention of neurodegeneration …
Modulation of sestrin confers protection to Cr(VI) induced neuronal cell death in Drosophila melanogaster.
PS Pallavi Singh, DK Chowdhuri - 2018 - cabidigitallibrary.org
Increased oxidative stress is one of the major causes of hexavalent chromium [Cr (VI)], a
heavy metal with diverse applications and environmental presence, induced neuronal
adversities in exposed organism including Drosophila. Sestrin (sesn), an oxidative stress
responsive gene, emerges as a novel player in the management of oxidative stress
response. It is reported to be regulated by Target of rapamycin (TOR) and the former
regulates autophagy and plays an important role in the prevention of neurodegeneration …
heavy metal with diverse applications and environmental presence, induced neuronal
adversities in exposed organism including Drosophila. Sestrin (sesn), an oxidative stress
responsive gene, emerges as a novel player in the management of oxidative stress
response. It is reported to be regulated by Target of rapamycin (TOR) and the former
regulates autophagy and plays an important role in the prevention of neurodegeneration …
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