Epigenetic theory of aging https://lnkd.in/gGnQAPNi
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New epigenetic clocks reinvent how we measure age https://lnkd.in/eE4hvJse?
New epigenetic clocks reinvent how we measure age
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New epigenetic clocks reinvent how we measure age https://lnkd.in/eE4hvJse?
New epigenetic clocks reinvent how we measure age
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New epigenetic clocks reinvent how we measure age https://lnkd.in/eE4hvJse?
New epigenetic clocks reinvent how we measure age
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Reversing Aging? Have you ever been curious about the potential secrets encapsulated in our epigenome that may hold the potential to decelerate the aging process? A pioneering review presents the Information Theory of Aging (ITOA), suggesting that epigenetic reprogramming may serve as a pivotal factor in reversing the effects of aging. This study uncovers how the disturbances in the epigenome could be responsible for aging and how this epigenetic 'noise' could potentially hasten the process. Investigating the malleability of aging and the epigenome, this review immerses itself into the fascinating territory of epigenetic loss during aging. Unlock the potential of epigenetic reprogramming in mitigating age-related information loss, illuminating insights into DNA demethylation and the crucial role of specific transcription factors in this rejuvenating process. Join the discourse on deciphering the mysteries enveloped in the epigenome and establishing a path for more precise approaches to rejuvenating tissues and reversing the aging process. https://lnkd.in/gHfYMFAF #Aging #Epigenetics #RejuvenationResearch
Can epigenetic reprogramming reverse aging?
https://longevity.technology
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New epigenetic clocks reinvent how we measure age https://lnkd.in/erXTQqrE?
New epigenetic clocks reinvent how we measure age
medicalxpress.com
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New epigenetic clocks reinvent how we measure age https://lnkd.in/eE4hvJse?
New epigenetic clocks reinvent how we measure age
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Quantum Thinker, Precision based personalization - Data + Systems + People, Biohacker, Traveler, Learning enthusiast, Reader, Sports & Fitness Lover
Could reframing #aging, #healthspan, & #longevity in terms of Information Theory of Aging (ITOA) unlock the secrets of #regeneration? When combined with advances in #technology/#AI what is possible?
Reversing Aging? Have you ever been curious about the potential secrets encapsulated in our epigenome that may hold the potential to decelerate the aging process? A pioneering review presents the Information Theory of Aging (ITOA), suggesting that epigenetic reprogramming may serve as a pivotal factor in reversing the effects of aging. This study uncovers how the disturbances in the epigenome could be responsible for aging and how this epigenetic 'noise' could potentially hasten the process. Investigating the malleability of aging and the epigenome, this review immerses itself into the fascinating territory of epigenetic loss during aging. Unlock the potential of epigenetic reprogramming in mitigating age-related information loss, illuminating insights into DNA demethylation and the crucial role of specific transcription factors in this rejuvenating process. Join the discourse on deciphering the mysteries enveloped in the epigenome and establishing a path for more precise approaches to rejuvenating tissues and reversing the aging process. https://lnkd.in/gHfYMFAF #Aging #Epigenetics #RejuvenationResearch
Can epigenetic reprogramming reverse aging?
https://longevity.technology
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Unraveling the complexities of anorexia, research underscores the pivotal roles played by genetics, hormones, and brain circuits in its development. 🧠🧬 Hormonal imbalances, genetic predispositions, and intricate brain circuitry intertwine to shape the path of anorexia. This profound insight highlights the multifaceted nature of the disorder and the need for a comprehensive understanding. #AnorexiaResearch #GeneticInfluence #HormonalFactors #BrainCircuitry #EatingDisorderInsights 📊✨ Unveiling these contributing factors provides a foundation for targeted interventions and holistic treatment approaches. By acknowledging the interplay of genetics, hormones, and brain circuits, the medical community takes a significant step toward personalized and effective anorexia management. 🌐💙 Understanding these intricate connections fosters compassion and advances efforts to support individuals affected by anorexia on their journey to recovery. #EatingDisorderAwareness #HolisticTreatment #ResearchBreakthroughs #MentalHealthInsights #CompassionateCare 🌈💪
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☑️ *READ DESCRIPTION BELOW:* Energy metabolic disorder and inflammation of microglia in Alzheimer’s Disease in response to therapy. When exposed to the AD environment, microglia polarizing to pro-inflammatory ‘M1’ phenotypes release many inflammatory cytokines. Microglia activate TLRs, which, in turn activate the TREM2/AKT/mTOR signaling pathway, leading to the upregulation of HIF-1α, GLUT, and PKM2. This results in reduced OXPHOS, leading to the production of reactive oxygen species (ROS) and inflammatory factors. Furthermore, the TREM2/AKT/mTOR and TLR4/NF-κB signaling pathways directly activate NLRP3, promoting inflammatory gene expression and further enhancing glycolysis. The drugs indicated in yellow text boxes act through target genes (in red) to induce anti-inflammatory responses and regulate microglial polarization and energy metabolic disorder, ultimately aiming to alleviate or treat AD. https://lnkd.in/er9K6wJm #Gesundheit #Bildung #Fuehrung #Coaching #Mindset #Motivation #Gehirn #Neuroscience #Psychologie #Persoenlichkeitsentwicklung #Kindheit #KeyNoteSpeaker #Humangenetik #Biochemie #Neuroleadership #Ernaehrung #Transformation #Stress #Demografie #Gender #Age #interkulturelleKompetenz #Epigenetik #Veraenderung #EmotionaleIntelligenz #Change #Gesellschaft #Organisationsentwicklung #Philosophie #Beratung # Quantum
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Although considered a rare disorder, #fragileX syndrome is the most common genetic cause of intellectual disability in the world. Fragile X patients can have a range of mild to severe intellectual disability with the potential for other conditions such as autism, delayed motor development, hyperactivity, behavioral problems and seizures. And while it’s well-known that fragile X is caused by the FMR1 gene, it’s less understood how the disorder physically affects brain development and function. Christopher Patzke, the John M. and Mary Jo Boler Assistant Professor of Biological Sciences, is collaborating with fragile X patients and families to study the disorder. Read more: https://bit.ly/3SACNXb Notre Dame Research #raredisease #rarediseaseresearch #notredamescience #fragilexsyndrome
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