Did you miss this Symposium, or would you like to watch the discussions and presentations again? Here is the link to the AAMDSIF YouTube Playlist for the 2024 Bone Marrow Failure Disease Scientific Symposium: https://lnkd.in/edgz34_m Watch now or save to watch later!
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Detecting glaucoma early is tough due to the lack of sensitive disease endpoints. At ARVO this year, Tiffany Heaster-Ford et al. authored an abstract that highlights subcellular dysfunction as an early marker of retinal ganglion cell (RGC) degeneration. Results: - Significant rise in mitochondrial ROS shortly after injury - Spectroscopic marker for mitochondrial dysfunction colocalizes with mitochondrial mass markers by 8 hours - Modest differences in mass and motility observed by 24 hours Key Findings: - Mitochondrial dysfunction precedes axon loss and visual decline - Elevated ROS in mitochondria is observed 4-24 hours post-injury - Changes in mitochondrial structure and motility detected within 24 hours Justin Elstrott, Pooja Teotia, Carmela Villegas, Marion Jeanne
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Learn more about NHLBI’s legacy of research that has advanced the understanding of #SickleCellDisease , improved clinical progress, & paved the way for a cure for all patients: Download Sickle Cell Disease: Milestones in Research in Clinical Progress: https://go.nih.gov/eVBJ8a0
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Curious about Neuroscience’s latest cover? Learn more about it here and the role of HSP90 in orchestrating different regulated cell death (RCD) mechanisms in Parkinson’s disease. 👉 Mansour et al, 2024: https://ow.ly/F0bX50QxcI6 👉 To the full issue: https://ow.ly/bcnm50QxcI3
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Unraveling the Mechanisms of Valvular Heart Disease to Identify Medical Therapy Targets: A Scientific Statement From the American Heart Association. This statement focuses on the most common primary valvular heart diseases, including calcific aortic stenosis, bicuspid aortic valves, mitral valve prolapse and rheumatic heart disease, and outlines the molecular discoveries contributing to each, potential therapeutic targets, and key areas of research priorities. http://spr.ly/60425ag8K
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Aging research is a field that concerns all of us. The new ReALity - funded Innovation Projects are therefore important, but also very interesting. If you would like to learn more, go to our website: reality.uni-mainz.de #researchrocks
What role does the endothelium play when it comes to vascular and neuronal inflammation, degeneration and cardiovascular disease? This is only one of the important questions, PI’s (Primary Investigators) are currently asking themselves as they have begun their new ReALity funded research project EndoAge. #research #innovationproject #lifesciences #agingresearch Photo: Kenny Eliason on Unsplash
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NEW ISSUE OUT 🔉 📌 A minifocus on interventions for peripheral artery disease; 📌 Imaging substudy from the BIOMAG-I first-in-human study; 📌 Lesion morphology and stent elongation during bifurcation PCI; 📌 debating the pros and cons of IVUS vs OCT for PCI guidance; and more Don't miss it 👉 https://lnkd.in/eQkyKhAe
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Interesting case of a thymoma found after the clinical diagnosis of rippling muscle disease. This highlights the importance of a thorough clinical examination in benign appearing symptoms such as cramps. With videos! https://lnkd.in/eJ7vKiMA
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❤️🧬If a picture is worth 1000 words, this animated PowerPoint presentation by Thomas Dayspring, MD FACP, FNLA for the Family Heart Foundation Lp(a) Support Group, which I had the pleasure of hosting last Monday evening, was worthy of a gold-medal. 🥇 Do you #KnowLpa? I in 5 people globally are born with this genetic risk for atherosclerosis and coronary vascular disease. Please click on the link to view the presentation (1 hour duration) and then get tested! https://lnkd.in/egr9W4_u
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A little bit of my Duke's Neuromuscular Disorder background: Arguments debunking synucleinopathy hypothesis, which proposes that Lewy pathology causes Parkinson's disease: - Aggregated α-synuclein in the form of Lewy bodies is not a space-occupying lesion but the insoluble fraction of its precursor, soluble monomeric α-synuclein; - Low cerebrospinal fluid α-synuclein levels predict brain atrophy and clinical disease progression. The transformation of α-synuclein into Lewy pathology may occur as a response to biological, toxic, or infectious stressors whose persistence perpetuates the nucleation process, depleting normal α-synuclein and eventually leading to Parkinson's symptoms from neuronal death. The loss-of-function synucleinopenia hypothesis by evaluating the clinical and neurodegenerative rescue effect of replenishing the levels of monomeric α-synuclein. 🧠 #neuromusuclardisorders #neuroscience
Professor of Neurology, Advocate of precision medicine in neurodegenerative diseases, co-founder of Regain Therapeutics, evaluating treatments to correct proteinopenia.
Synucleinopenia and the Origin of Parkinson's Disease --the final and unrestricted version just published. https://lnkd.in/gfC3PsC4
Loss of monomeric alpha-synuclein (synucleinopenia) and the origin of Parkinson's disease
sciencedirect.com
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Happy #NationalDNAday! Learn how #MDA has used #DNA to further neuromuscular disease research from MDA President & CEO, Donald S. Wood, PhD. Read more about our commitment to breakthroughs in research at MDA.org/science
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